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The small amount of increased oxygen that is needed by the cells to improve their function does not necessarily require new blood vessel formation to be accomplished because with HBOT free oxygen molecules are dissolved directly into the cerebrospinal fluid Therefore these cells are no longer dependent on increased oxygen delivery from increased blood vessel formation that secondarily delivers a greater oxygen load because it carries more hemoglobin.

At times it may actually be the sudden removal of higher than normal oxygen concentrations that the body has adapted to rather than the higher levels of oxygen itself that may stimulate angioneogenesis There are reports that the new vessel formation in the retinas of premature infants who were on high doses of oxygen was stimulated by the rapid removal of oxygen and not from the oxygen itself.

Efrain Olszewer has pre- and post-angiograms documenting collateral circulation beginning as early as ten to twenty hours after initiating hyperbaric therapy for cerebral vascular disease and peripheral arteriosclerosis at pressures lower than 1.3 ATA It is known that one of the problems children with autism have is decreased blood flow to the brain (cerebral hypoperfusion).

Therefore is has been speculated that angioneogenesis is the way that HBOT helps autism However, though angioneogenesis may be one mechanism by which children with autism are helped by HBOT, angioneogenesis may not be the primary mechanism by which HBOT works The amount of cerebral hypoperfusion in autistics compared to controls is about 8%, so a small increase in oxygen delivery may be all that is needed to overcome this deficit and show clinical benefit.

Recent studies have demonstrated that children with autism frequently have neuro-inflammatory and gastrointestinal inflammatory conditions occurring.

Multiple studies demonstrate the beneficial effect of hyperbaric oxygen therapy in inflammatory conditions.

Likewise it is my theory that it only takes a little bit of increased oxygen tension for viruses to leave.

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HBOT may have the potential to activate dysfunctional mitochondria and/or to activate "dormant/idling cells" thereby allowing more "mitochondrial product" to be appreciated by the body.However, as history has shown repeatedly throughout the years, convention is only convention until challenged, proven wrong, and then changed. A few of the multiple mechanisms demonstrating how HBOT may work for children with autism was originally and thoroughly researched over many months time by Dr. These mechanisms are shown below along with a couple others that have been suggested They include: The growth of new blood vessels has been shown to occur from soft chambers as well as from hard, and is a process that may continue to increase subsequent to discontinuing therapy for a period of time after oxygen loading.Though it has been stated often on the internet boards that angioneogenesis does not occur unless pressures are 1.5 ATA or greater, the South American physician Dr., not only due to the competing mechanisms of vasodilation and vasoconstriction, but also due to decreasing the inflammation that secondarily constricts blood vessel lumens in a closed spac.Because inflammation is accompanied by swelling, tissue expansion or compression will occur.

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